Birth
Control - The Silent Abortion
Different types of birth prevention pills (oral contraceptive), and how they work.
Oral Contraceptives (OCs).
(1) Suppression of Ovulation. When the female reproductive
system is functioning normally, the hypothalamus (the
part of
the brain containing the vital autonomic regulatory centers)
controls the release of gonadotropin-releasing hormone
(GnRH),
which signals the pituitary gland to secrete luteinizing
hormone
(LH), which in turn assists ovulation and coordinates
the
release of estrogen and progestin from the ovaries.
When a woman ingests birth prevention pills, they literally
hijack her reproductive system. The pills cause the ovaries
to maintain a steady high level of estrogen and/or progestin
production, depending upon the type and brand of pill
being
used. Thus, the woman's body is essentially "tricked"
into
acting as if it is continuously pregnant. The hypothalamus
adjusts to this high level of hormone secretion and essentially
shuts off GnRH production.
Therefore, the production of luteinizing hormone by the
pituitary gland is also inhibited, and ovulation either
ceases
or is drastically curtailed. In those months that ovulation
is
suppressed, the mode of action of the birth prevention
pill
is contraceptive in nature.
(2) Cervical Mucus Effects. Oral contraceptives also cause
changes in the consistency and acidity of cervical mucus,
making it more difficult for sperm to penetrate and live
in
the cervix, a second contraceptive effect.
(3) Endometrial Effects. The third effect of oral contraceptives
on
the body is to cause certain changes in the endometrium
(lining of the uterus), making implantation more difficult.
In a
cycle where ovulation was not prevented and fertilization
takes
place, the
pill causes a "silent abortion."
Two-Fold vs. Three-Fold Action.
Most of the older "high-dose" birth prevention pills functioned
mainly by inhibiting ovulation and affecting the cervical
mucus,
making them primarily two-fold in function.
Sometimes, however, breakthrough ovulation occurred, and
so
the older pills were occasionally
abortifacient in their
actions.
All of the newer birth prevention pills on the market
today not
only often suppress ovulation and affect the cervical
mucus,
they often make implantation of the developing human
being
impossible. This three-fold mode of function means that
all
of the newer birth prevention pills function at
least part of the time as abortifacients.
The Types of Oral Contraceptive.
Overview.
There are currently three general classes of birth prevention
pill
manufactured in the United States and other countries
and used
worldwide. These are the high-dose pill, the low-dose
pill, and
the "mini-pill." The following paragraphs describe these
pills and
their modes of action.
The High-Dose Pill.
The Searle Pharmaceutical Corporation developed the first
oral
contraceptive, Enovid, in the late 1950s. In keeping
with its
defensive anti-lawsuit strategy, the company tested the
pill on
Puerto Rican women before concluding in 1961 that it
was safe
for women on the American mainland to use. 1
1 Bogomir M. Kuhar, Ph.D. "Pharmaceutical Companies: The
New Abortionists."
Reprint 16 from Human Life International, 4 Family Life,
Front Royal, Virginia 22630.
Experimentation on foreign women is a typical tactic of
the
major pharmaceutical companies. They often test birth
prevention chemicals and devices on poor women in developing
countries so any mistakes or serious health problems
are easier
to cover up. Poor women on foreign countries have little
recourse
when their health is destroyed or damaged by this kind
of testing.
This is because the companies bring huge amounts of money
to
their homelands, and protest against the programs can
easily be
suppressed by local or national governments.
Enovid and other high-dose pills, which have generally
fallen out
of favor in the United States but are still widely used
in developing
countries, contained from 1 to 12 milligrams of progestin
and/or 60
to 120 micrograms of estrogen, a natural female hormone.
This high
dosage had a variety of effects, including blurred vision,
nausea,
weight gain, painful breasts, cramping, irregular menstrual
bleeding,
headaches, and possibly breast cancer.1
1 Bogomir M. Kuhar, Ph.D. "Pharmaceutical Companies: The
New Abortionists."
Reprint 16 from Human Life International, 4 Family Life,
Front Royal, Virginia 22630.
The high-dose pills were primarily two-fold in action.
Their
primary mechanism suppressed gonadotropin production
and
therefore ovulation. They also caused changes in the
consistency
and acidity of cervical mucus, making it more difficult
for sperm
to penetrate and live in the cervix. Finally, they occasionally
caused certain changes in the endometrium (lining of
the uterus),
making implantation more difficult.
When the high-dose pill functioned by this last mechanism,
it was
an abortifacient if the woman experienced a "breakthrough"
ovulation. Although this occurred only during about 1
to
12 percent of all cycles, it was not the primary intent
of the
manufacturers.
Beginning about 1975, pill makers, in reaction to bad
publicity
about the severe side effects of the high-dosage pills,
steadily
decreased the content of estrogen and progestin in their
products.
The Low-Dose Pill.
Eventually, the older "high-dose" pills gave way to the
new,
abortifacient "low-dose" pills. Ortho/Johnson & Johnson,
G.D. Searle/Monsanto, and Syntex, the three largest manufacturers
of OCs in the United States, voluntarily withdrew their
"high-dose"
products from the U.S. market in 1988 on the advice of
the U.S.
Food and Drug Administration (FDA). These were the last
commercially-available pills containing more than 50
micrograms
of estrogen. 1
1 Bogomir M. Kuhar, Ph.D. "Pharmaceutical Companies:
The New Abortionists."
Reprint 16 from Human Life International, 4 Family Life,
Front Royal, Virginia 22630.
The low-dose pills contain from 0.35 to 15 milligrams
of progestin
in the form of norethindrone, norgestrel, ethyndiol diacetate,
or
norethindrone acetate, and from 0.7 to 2.0 micrograms
of
estrogen in the form of ethinyl estradiol or mestranol,
a tremendous
drop in estrogenic potency compared to the high-dose
pills. 2
2 Robert A. Hatcher, et. al. Contraceptive Technology
(17th Revised Edition).
New York: Ardent Media, Inc., 1998. Table 19-1, "Relative
Potency of Estrogens
and Progestins in Currently Available Oral Contraceptives
Reflecting the Debate
About the Strength of the Progestins," page 407.
The low-dose pills work in essentially the same manner
as the
high-dose pill. However, a much higher percentage of
ovulation
occurs in women who use the low-dose pills, due to the
much lower
estrogen dose. This means that women who use these
pills frequently
conceive, and the low-dose pills prevent implantation
of the new
human life, thereby acting more often as true
abortifacients.
The Mini-Pill.
Scientists have not pinpointed the primary mechanism of
mini-pills
(progestin-only pills), although women who use them frequently
ovulate. Therefore, these pills function primarily as
abortifacients.
It is known that pills that contain only progestin alter
the cervical
mucus. They also interfere with implantation by affecting
the
endometrium (lining of the uterus) and suppressing ovulation
in
some women by reducing the presence of follicle-stimulating
hormone (FSH).
This mechanism is confirmed by the Food and Drug Administration,
which has stated that "Progestin-only contraceptives
are known to
alter the cervical mucus, exert a progestinal effect
on the endometrium,
interfere with implantation, and, in some patients, suppress
ovulation." 3
3 Federal Register, 41:236, December 7, 1976, page 53,634.
The Department of Health and Human Services (HHS), in
its
1984 pamphlet "Facts About Oral Contraceptives," compared
the
action of high-dose pills and mini-pills:
It is possible for women using
combined pills (synthetic estrogen
and progestin) to ovulate. Then other
mechanisms work to prevent
pregnancy. Both kinds of pills make
the cervical mucus thick and
"inhospitable" to sperm, discouraging
any entry to the uterus. In
addition, they make it difficult for
a fertilized egg to implant, by
causing changes in Fallopian tube
contractions and in the uterine
lining. These actions explain why
the minipill works, as it generally
does not suppress ovulation
[emphasis added].
The makers of the mini-pills also admit this mode
of action. For
example, Syntex Laboratories announced that its progestin-only
pill Norinyl "... did not interfere with ovulation ...
It seems
to affect the endometrium so that a fertilized egg cannot
be
implanted." 4
4 United Press International news release in the Cincinnati Post, January 11, 1973.
In other words, the pill is now truly abortifacient "birth
prevention"
-- not conception control, as may have originally been
intended
when the first oral contraceptives were being developed.
"Emergency Contraceptives" - morning-after pills.
For 30 years, pro-abortionists and "family planners" have
searched
for new and better ways to kill early preborn babies
with chemical
compounds and devices already approved by the FDA. The
methotrexate/misoprostol combination described above
is one of
the results of this ongoing search.
Anti-lifers have also promoted another type of abortifacient
potion,
the "Yuzpe Regimen," which consists of women taking combined
ethinyl estradiol/levonorgestrel pills at a higher than
normal dose.
This kind of abortifacient is ideal for women who are
forgetful or
lazy about taking their pills. 5
5 "What are Legalities of Promoting ECPs?" Contraceptive
Technology Update,
November 1995, pages 137 and 138.
Pro-abortionists dishonestly call this "emergency contraception,"
another attempt to erase the distinction between true
contraception
and abortifacient action. Pills taken under the Yuzpe
and similar
regimens are often called "emergency contraceptive pills
(ECPs),"
"morning-after pills (MAPs)," and "postcoital contraception."
When pro-lifers hear pro-abortionists using these and
similar
terms, they can be sure that they are referring to abortifacient
cocktails.
Pregnancies While Using the "Infallible" Pill.
From the very first day that it was introduced, the oral
contraceptive
has been hailed as the solution to "unwanted pregnancies"
and the
enabler of the Sexual Revolution. Continued allegations
of high
efficiency, combined with the easy availability of abortion
as a
"backup," have inevitably led to widespread careless
use of the
pill.
Only about 11 percent of all women who use the pill do
so correctly,
according to a 1989 study. 6
6 Kim Painter. "Most Users of the Pill Don't Follow Directions."
USA Today,
February 21, 1990, page D1.
This carelessness is the major contributor to an incredible
number
of unintended pregnancies, especially among younger women.
There are about 630,000 pregnancies annually among U.S.
women
who are on the pill, and more than 80 percent of these
occur among
women 15 to 24 years old.
Among women in this age group, the method effectiveness
of the
birth prevention pill is 96.2 percent per year, significantly
lower than
the effectiveness for older women. This percentage still
sounds very
high indeed; but the method effectiveness refers to the
efficiency of
the pill when a woman is in very good health and uses
the pill without
error. When user error is factored in, the result is
the actual user
effectiveness rate, also known as the overall effectiveness
rate. 6
6 Kim Painter. "Most Users of the Pill Don't Follow Directions."
USA Today,
February 21, 1990, page D1.
The overall effectiveness rate for the low-dose pill is
89 percent
per year. 6
6 Kim Painter. "Most Users of the Pill Don't Follow Directions."
USA Today,
February 21, 1990, page D1.
This still sounds high until you calculate the probability
of a woman
15 to 24 years old becoming pregnant over an extended
period of time
when using the pill.
In summary, if a fornicating girl of 15 starts using the
pill, and
uses it continuously, there is a better than 50 percent
chance that
she will become pregnant by the time she is 22!
This statistic is verified by pro-abortionists, including
Planned
Parenthood abortion statistician Dr. Christopher Tietze,
who said
that "within 10 years, 20 to 50 percent of pill users
and a substantial
majority of users of other methods may be expected to
experience at
least one repeat abortion." 7
7 Christopher Tietze, quoted in the National Abortion
Rights Action League's
A Speaker's and Debater's Guidebook. June 1978, page
24.
Note that Tietze is speaking about repeat (second or more)
abortions
here. These statistics are significant when one considers
that one of
the primary goals of school-based clinics (SBCs) is to
distribute
contraceptives and abortifacients to teenagers without
parental
consent or knowledge.
Implications for Pro-Life Activists.
Millions of women in the USA and all over the world use
oral
contraceptives. Many women who would never even consider
a surgical abortion now use low-dose birth prevention
pills
that cause them to abort a new life an average of once
or twice
every year. A large number of women who say that they
are
pro-life use these pills. Many at the urging of their
husbands.
These are usually the women who are ignorant
of the pill's
abortifacient mode of action,
those who think that their way
of life requires that they use the pill, or those who
cannot mentally
make the connection between contraception and abortion.
Some researchers (using very conservative figures) have
calculated
that the birth prevention pill directly causes between
1.53 and 4.15
million chemical abortions per year in the United States
-- up to two
and a half times the total number of surgical abortions
committed
every year! 8
8 S. Killick, E. Eyong, and M. Elstein. "Ovarian Follicular
Development in Oral Contraceptive
Cycles." Fertility and Sterility, September 1987, pages
409 to 413.
This means that "pro-life" women who are using an oral
contraceptive
or some other means of abortifacient birth control are
committing
abortions themselves on a frequent basis. These abortions
are "silent"
and unseen, but they are no less abortions in the eyes
of God than are
gruesome third-trimester D&X abortions. There are
many "pro-lifers"
who are using these pills and who are involved in their
promotion and
distribution. These people must consider whether they
can, in good
conscience, criticize women whose action differs from
their own
only in that they have to drive to an clinic mill to
commit it.
1 Bogomir M. Kuhar, Ph.D. "Pharmaceutical Companies: The
New Abortionists." Reprint 16 from Human Life
International, 4 Family Life, Front Royal, Virginia 22630.
2 Robert A. Hatcher, et. al. Contraceptive Technology
(17th Revised Edition). New York: Ardent Media, Inc., 1998.
Table 19-1, "Relative Potency of Estrogens and Progestins
in Currently Available Oral Contraceptives Reflecting the Debate
About the Strength of the Progestins," page 407.
3 Federal Register, 41:236, December 7, 1976, page 53,634.
4 United Press International news release in the Cincinnati
Post, January 11, 1973.
5 "What are Legalities of Promoting ECPs?" Contraceptive
Technology Update, November 1995, pages 137 and 138.
6 Kim Painter. "Most Users of the Pill Don't Follow Directions."
USA Today, February 21, 1990, page D1.
7 Christopher Tietze, quoted in the National Abortion
Rights Action League's A Speaker's and Debater's Guidebook.
June 1978, page 24.
8 S. Killick, E. Eyong, and M. Elstein. "Ovarian Follicular
Development in Oral Contraceptive Cycles." Fertility and
Sterility, September 1987, pages 409 to 413.
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